Document Type

Article

Publication Date

6-1-2017

Publication Title

Bioorganic and Medicinal Chemistry Letters

Abstract

Copalic acid, one of the diterpenoid acids in copaiba oil, inhibited the chaperone function of α-crystallin and heat shock protein 27 kD (HSP27). It also showed potent activity in decreasing an HSP27 client protein, androgen receptor (AR), which makes it useful in prostate cancer treatment or prevention. To develop potent drug candidates to decrease the AR level in prostate cancer cells, more copalic acid analogs were synthesized. Using the level of AR as the readout, 15 of the copalic acid analogs were screened and two compounds were much more potent than copalic acid. The compounds also dose-dependently inhibited AR positive prostate cancer cell growth. Furthermore, they inhibited the chaperone activity of α-crystallin as well.

Comments

This work was supported by Center for Gene Regulation in Health and Disease (GRHD) and summer undergraduate research program of Cleveland State University. Aicha Quamine was supported by McNair scholarship.

DOI

10.1016/j.bmcl.2017.04.046

Version

Postprint

Volume

27

Issue

11

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